Clot formation
Thrombosis — Pathological Clot Formation
Also searched as Thrombosis — Pathological Clot Formation.
You want it low: a clot inside a living vessel serves no purpose and ends organs.
In depth
Thrombosis — a clot forming inside a vessel that should have stayed open, and the occlusion that follows.
What drives it
Reduces clotting increases it.
The fast arm. Verbatim: 'Arterial thrombi are rich in platelets and form at the sides of or around ruptured atherosclerotic plaques' (Eur Heart J, PMID 28039338). Arterial only — venous thrombi are fibrin- and red-cell-rich. PMID 28039338
Clotting increases it.
The mesh that sets the clot. Verbatim: 'Fibrin clot formation is the ultimate common event in the coagulation cascade leading to thrombus formation' (PMC7867291). Cascade topology, not an intervention effect size. PMC7867291
Clot breakdown reduces it.
The counter-force. Verbatim: 'Endogenous fibrinolysis is an important physiological countermeasure against development and lasting arterial or venous thrombosis' (PMC7867291). Physiological — laboratory measures, not a supplement trial. PMC7867291
What it drives
It reduces Heart & blood pressure.
Why it matters. Verbatim: 'Arterial thrombosis is the cause of myocardial infarction (MI) and stroke, while venous thrombosis (VT) leads to venous thromboembolism (VTE) and pulmonary embolism (PE)' (Eur Heart J, PMID 28039338). Attribution, no quantified risk. PMID 28039338
Blends that modulate it
Each blend page names this pathway and carries the study behind its claim.
See it on the map
Open the causal map to follow this pathway upstream and downstream, arrow by arrow, each with its study.