Abdominal fluid
Portal hypertensive ascites↓
Also searched as Portal hypertensive ascites.
You want it never to appear. Its first appearance is the moment compensated becomes decompensated.
In depth
Fluid collecting in the abdomen because pressure in the portal system forces it out of the circulation. It is the most common first decompensating event, and its arrival changes the prognosis sharply.
What drives it
Portal pressure increases it.
Verbatim (pmc11945444): 'ascites is the most common complication from cirrhosis related to portal hypertension and depicts the onset of hepatic decompensation. Ranging from uncomplicated to refractory ascites, the progression carries prognostic value by reflecting the deterioration of underlying cirrhosis and portal hypertension.' scope: a treatment hazard is named in the same source — 'with worsening systemic vasodilation and inflammation when ascites progresses, patients on NSBBs are at risk of hemodynamic collapse leading to renal hypoperfusion and thus hepatorenal syndrome.' not a node here, but named.
What it drives
It increases Liver decompensation.
Verbatim (pmc12453315, on the Baveno vii shift): 'this paradigm shifted from solely preventing variceal bleeding to preventing hepatic decompensation more broadly. This approach redefines decompensation as a whole, recognizing that any initial decompensating event, especially ascites (the most common), significantly worsens patient prognosis and can be prevented with therapy.'.
Blends that modulate it
Each blend page names this pathway and carries the study behind its claim.
See it on the map
Open the causal map to follow this pathway upstream and downstream, arrow by arrow, each with its study.