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Bloodwork

Albumin

Reads: Blood protein status

The Ferry

1 formula on this site modulates this pathway.

You want enough of it arriving. Fatty acids do not dissolve in blood; they travel bound. When carrier supply was varied in perfused tissue while release was held constant, NET output still changed — proof that carriage, not the enzymes, can be the limiting step. The reciprocal is measured too: scavenging fatty acids away from the cell with extra carrier prevents the feedback inhibition that free fatty acids otherwise exert on their own release. Carry them off and the brake never engages.

What this marker reads

The blood protein that carries released fat away — without it, fat cannot leave the tissue.

What a named body publishes

Each line below is the sentence the issuing body wrote, carried with its own identifier. Nothing here interprets your result — it states what was published, for whom, and by whom.

Albumin

“Serum albumin, ages 18-39: 36-48 g/L, from the Nordic Reference Interval Project; the published table carries no sex stratification for albumin.” 18-39

Nya gemensamma nordiska referensintervall inom klinisk kemi · Läkartidningen 2004;101:901-5 The document

“Serum albumin, ages 40-69: 36-45 g/L.” 40-69

Nya gemensamma nordiska referensintervall inom klinisk kemi · Läkartidningen 2004;101:901-5 The document

“Serum albumin, ages 70 and over: 34-45 g/L.” 70+

Nya gemensamma nordiska referensintervall inom klinisk kemi · Läkartidningen 2004;101:901-5 The document

Your laboratory reads your result against its own assay, and this page does not do that for it. What is above is what a named body published and for whom; what is below is the machinery the marker reads and the formulas witnessed on it.

What moves it

Liver raises it.

Albumin is synthesized exclusively by hepatocytes; serum albumin is the classical index of hepatic synthetic function. PMID 29295548

Liver scarring lowers it.

Loss of functional hepatocyte mass in cirrhosis reduces albumin synthesis and lowers serum albumin. doi 10.22416/1382-4376-2022-32-4-7-16

What it moves

It raises Blood flow to fat tissue.

'The supply of free fatty acid (ffa) carrier to the tissue was varied either by variations in the rate of blood flow OR by changes in the albumin concentration of the perfusing blood at constant flow rate. The net production of FFA from the tissue was found to depend on the supply of carrier' (PMID 7195565). ⇒ flow and carrier are two ways of delivering the same thing. PMID 7195565

It lowers Fat re-storage.

The reciprocal test, and it is the strongest evidence on this pathway. 'The inhibition of FFAs on lipolysis is associated with a reduced production of cAMP due to AC inhibition, which can be prevented by reducing intracellular ffa level via extracellular albumin scavenging' (doi 10.3389/fphys.2022.826314). Carry the fatty acids off and the feedback brake never engages. doi 10.3389/fphys.2022.826314

Formulas that modulate it

Each formula page names this pathway and carries the study behind its claim.

Lipolytic (Stored-Fat Mobilization) →

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